This profile is built from public research funding records (CIHR, NSERC and SSHRC) and PubMed. We have not imported them from a University of Ottawa directory, so their courses and email address may be missing. Find their university profile.
Research
Latest papers
Beta-Cell-Specific Expression of Nicotinamide Adenine Dinucleotide Phosphate Oxidase 5 Aggravates High-Fat Diet-Induced Impairment of Islet Insulin Secretion in Mice.
Antioxidants & redox signaling · 2020
NADPH Oxidase 5 Is a Pro-Contractile Nox Isoform and a Point of Cross-Talk for Calcium and Redox Signaling-Implications in Vascular Function.
Journal of the American Heart Association · 2018
Protein Kinase C-δ Mediates Shedding of Angiotensin-Converting Enzyme 2 from Proximal Tubular Cells.
Frontiers in pharmacology · 2016
Latest funding
- $15,000
Canadian Kidney Discoveries Network Meeting 2026
CIHR · 2025 · Nominated PI
- $100,000
The role of G protein receptor 84 in inflammatory kidney injury
CIHR · 2024 · Nominated PI
- $822,375
The role of Nox5 in hypertension-associated renal injury
CIHR · 2018 · Nominated PI
4 publications.
Beta-Cell-Specific Expression of Nicotinamide Adenine Dinucleotide Phosphate Oxidase 5 Aggravates High-Fat Diet-Induced Impairment of Islet Insulin Secretion in Mice.
Bouzakri K, Veyrat-Durebex C, Holterman C, Arous C, Barbieux C, Bosco D, Altirriba J, Alibashe M, Tournier BB, Gunton JE, Mouche S, Bietiger W, Forterre A, Berney T, Pinget M, Christofori G, Kennedy C, Szanto I
NADPH Oxidase 5 Is a Pro-Contractile Nox Isoform and a Point of Cross-Talk for Calcium and Redox Signaling-Implications in Vascular Function.
Montezano AC, De Lucca Camargo L, Persson P, Rios FJ, Harvey AP, Anagnostopoulou A, Palacios R, Gandara ACP, Alves-Lopes R, Neves KB, Dulak-Lis M, Holterman CE, de Oliveira PL, Graham D, Kennedy C, Touyz RM
Protein Kinase C-δ Mediates Shedding of Angiotensin-Converting Enzyme 2 from Proximal Tubular Cells.
Xiao F, Zimpelmann J, Burger D, Kennedy C, Hébert RL, Burns KD
sirt1-null mice develop an autoimmune-like condition.
Sequeira J, Boily G, Bazinet S, Saliba S, He X, Jardine K, Kennedy C, Staines W, Rousseaux C, Mueller R, McBurney MW
Canadian Kidney Discoveries Network Meeting 2026
Principal investigators: Kennedy, Christopher R
Keywords: Capacity Building; Collaboration; Kidney Disease Research; Knowledge Mobilization; Learner Development; Networking
The role of G protein receptor 84 in inflammatory kidney injury
Principal investigators: Kennedy, Christopher R
Keywords: G Protein Receptor; Glomerulonephritis; Neutrophils; Podocyte
The role of Nox5 in hypertension-associated renal injury
Principal investigators: Kennedy, Christopher R
Keywords: Angiotensin Ii; Chronic Kidney Disease; Hypertension; Inflammation; Macrophages; Nox5; Oxidative Stress; Transgenic Mice
Nox5 derived reactive oxygen species in vascular and renal injury during hypertension
Principal investigators: Majane, Olebogeng H
Keywords: Hypertension; Oxidative Stress; Signal Transduction; Vascular Biology
Validating Nox5 as a target for diabetic renal complications
Principal investigators: Kennedy, Christopher R
Keywords: Diabetes; Kidney; Podocyte; Reactive Oxygen Species; Transgenic Mice
Prostaglandin signaling and diabetic nephropathy: impact on podocyte health
Principal investigators: Thibodeau, Jean-Francois
Keywords: Cytoskeleton; Diabetic Nephropathy; Glomerular Disease; Immunofluorescence; Podocytes; Prostaglandins; Proteinuria; Rho/Rock Pathway; Stz Diabetic Mice Model; Transgenic
Nox homologues, redox signaling and vascular biology in hypertension
Principal investigators: Touyz, Rhian M
Keywords: Animal Physiology; Free Radical; Hypertension; Molecular And Cell Biology; Oxidative Stress; Remodeling
Prostanoid Signaling in Diabetic Nephropathy
Principal investigators: Kennedy, Christopher R
Keywords: Cell Signaling; Diabetic Nephropathy; Gene-Targeted Mice; Podocyte; Prostanoid Receptors; Proteinuria
From CIHR, NSERC and SSHRC funding decisions: CIHR since 2008, NSERC since 1991 and SSHRC since 1998, including their latest published competition results.
Frequent collaborators
- Cellular and Molecular Medicine
- Medicine
- Pathology and Laboratory Medicine
Co-authors at University of Ottawa, colored by department. Thicker lines mean more shared papers; select anyone to open their profile and their own map.
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