Faculty profile

Jane Mitchell

ResearcherPharmacology and ToxicologyUniversity of Toronto

This profile is built from public research funding records (CIHR, NSERC and SSHRC) and PubMed. We have not imported them from a University of Toronto directory, so their courses may be missing. Find their university profile.

Jane Mitchell

Researcher

Pharmacology and Toxicology

Health Sciences

University of Toronto

Toronto, Ontario, Canada

Supervision signals

Has led research grants, which requires an independent research appointment. Ask their department whether they supervise graduate students.
6 publications in the last five years, latest in 2026.
Senior (last) author on 5 recent papers, a sign of leading a research group.
Principal investigator on 11 research grants (6 CIHR, 5 NSERC), most recently in 2019.

These signals come from public records, including CIHR, NSERC and SSHRC funding decisions. Ask the professor or the graduate program whether they are taking new students.

Academic metrics

Publications

Collaborators

Grants

Publications by year

Counts cover the publications linked to this profile. Citations and the h-index appear when the source records them.

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Signals and details

Research

Exercise Menopause Bone Bone Fracture Healing Bone Strength G Proteins Signaling Sex-Specific Effects Of Exercise

Latest papers

Increased osteoblast Gα11 level compromises bone healing quality by suppressing high-density bone formation.
Bone · 2026 · senior author
Insulin-like growth factor-1 and cognition in normoglycemia, prediabetes, and type 2 diabetes mellitus.
Psychoneuroendocrinology · 2024
Increased Osteoblast GαS Promotes Ossification by Suppressing Cartilage and Enhancing Callus Mineralization During Fracture Repair in Mice.
JBMR plus · 2023 · senior author

All 37 publications

Latest funding

Pro-Resolving Inflammatory Mediators in Neurovascular Gains in Aerobic Training (PRIMiNG-AT)
CIHR · 2021 · Co-investigator
$747,439
Variation in G protein expression in bone cells determines bone quality and skeletal responses to catabolic and anabolic stimuli
CIHR · 2019 · Nominated PI
$100,000
Neurocognitive effects of osteocalcin in type 2 diabetes
CIHR · 2018 · Co-investigator
$933,300

All 15 grants and awards

37 publications.

Increased osteoblast Gα11 level compromises bone healing quality by suppressing high-density bone formation.

Lee KK, Changoor A, Grynpas MD, Mitchell J

Bone2026Senior author

DOI: 10.1016/j.bone.2026.117893 PMID: 41962787

Insulin-like growth factor-1 and cognition in normoglycemia, prediabetes, and type 2 diabetes mellitus.

Ryoo SW, Anita NZ, Perlman G, Xiong LY, Wu CY, Wood M, Rabin JS, Mitchell J, Swardfager W

Psychoneuroendocrinology2024

DOI: 10.1016/j.psyneuen.2023.106946 PMID: 38198904

Increased Osteoblast GαS Promotes Ossification by Suppressing Cartilage and Enhancing Callus Mineralization During Fracture Repair in Mice.

Lee KK, Changoor A, Grynpas MD, Mitchell J

JBMR plus2023Senior author

DOI: 10.1002/jbm4.10841 PMID: 38130768

β-Arrestin 2 knockout prevents bone loss in response to continuous parathyroid hormone stimulation in male and female mice.

Li Feng G, Grynpas MD, Mitchell J

Connective tissue research2023Senior author

DOI: 10.1080/03008207.2023.2199086 PMID: 37046359

Increased osteoblast GαS level determines bone response to hyperparathyroidism in female mice.

Zhang L, Lee KK, Sugamori KS, Grynpas MD, Mitchell J

The Journal of endocrinology2022Senior author

DOI: 10.1530/joe-22-0045 PMID: 35638565

Sex Differences in Metabolic and Behavioral Responses to Exercise but Not Exogenous Osteocalcin Treatment in Mice Fed a High Fat Diet.

Winberg J, Rentz J, Sugamori K, Swardfager W, Mitchell J

Frontiers in physiology2022Senior author

DOI: 10.3389/fphys.2022.831056 PMID: 35309065

Serum osteocalcin is associated with subjective stress in people with depression and type 2 diabetes.

Nguyen MM, Anita NZ, Darwish L, Major-Orfao C, Colby-Milley J, Wong SK, Sugamori KS, Lanctôt KL, Herrmann N, Oh PI, Yang P, Shah BR, Gilbert J, Assal A, Halperin IJ, Mitchell J, Swardfager W

Psychoneuroendocrinology2020

DOI: 10.1016/j.psyneuen.2020.104878 PMID: 33038647

Endostatin as a Mediator Between Endothelial Function and Cognitive Performance in Those at Risk for Vascular Cognitive Impairment.

Isaacs-Trepanier C, Saleem M, Herrmann N, Swardfager W, Oh PI, Goldstein BI, Mitchell J, Sugamori KS, Lanctôt KL

Journal of Alzheimer's disease : JAD2020

DOI: 10.3233/jad-200058 PMID: 32538839

Growth Hormone Increases Bone Toughness and Decreases Muscle Inflammation in Glucocorticoid-Treated Mdx Mice, Model of Duchenne Muscular Dystrophy.

Yoon SH, Grynpas MD, Mitchell J

Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research2019Senior author

DOI: 10.1002/jbmr.3718 PMID: 31188496

Intermittent PTH treatment improves bone and muscle in glucocorticoid treated Mdx mice: A model of Duchenne Muscular Dystrophy.

Yoon SH, Grynpas M, Mitchell J

Bone2019Senior author

DOI: 10.1016/j.bone.2019.01.028 PMID: 30716510

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Jane Mitchell

Research

Latest papers

Latest funding

Increased osteoblast Gα11 level compromises bone healing quality by suppressing high-density bone formation.

Insulin-like growth factor-1 and cognition in normoglycemia, prediabetes, and type 2 diabetes mellitus.

Increased Osteoblast GαS Promotes Ossification by Suppressing Cartilage and Enhancing Callus Mineralization During Fracture Repair in Mice.

β-Arrestin 2 knockout prevents bone loss in response to continuous parathyroid hormone stimulation in male and female mice.

Increased osteoblast GαS level determines bone response to hyperparathyroidism in female mice.

Sex Differences in Metabolic and Behavioral Responses to Exercise but Not Exogenous Osteocalcin Treatment in Mice Fed a High Fat Diet.

Serum osteocalcin is associated with subjective stress in people with depression and type 2 diabetes.

Endostatin as a Mediator Between Endothelial Function and Cognitive Performance in Those at Risk for Vascular Cognitive Impairment.

Growth Hormone Increases Bone Toughness and Decreases Muscle Inflammation in Glucocorticoid-Treated Mdx Mice, Model of Duchenne Muscular Dystrophy.

Intermittent PTH treatment improves bone and muscle in glucocorticoid treated Mdx mice: A model of Duchenne Muscular Dystrophy.

Pro-Resolving Inflammatory Mediators in Neurovascular Gains in Aerobic Training (PRIMiNG-AT)

Variation in G protein expression in bone cells determines bone quality and skeletal responses to catabolic and anabolic stimuli

Neurocognitive effects of osteocalcin in type 2 diabetes

Sex Differences in Bone Osteocalcin Response to Exercise

Variation in G protein expression in bone cells determines bone quality and response to catabolic and anabolic stimuli

Growth arrest and osteoporosis in Duchenne muscular dystrophy patients treated with glucocorticoids

Glucocorticoid Induced Osteoporosis (GIO) in the Paediatric Patient with Chronic Disease: Pathophysiology, Prediction and Management.

Glucocorticoid-induced osteoporosis: the role of G protein upregulation in osteoblasts

Glucocorticoid-induced osteoporosis: the role of G11 overexpression in osteoblasts

Molecular mechanisms of invertebrate visual systems

Frequent collaborators

Walter Swardfager

Adele Changoor

Marc Grynpas

Nathan Herrmann

Jennifer Harrington

Jeremy Gilbert

Jane Mitchell

Research

Latest papers

Latest funding

Increased osteoblast G&#x3b1;11 level compromises bone healing quality by suppressing high-density bone formation.

Insulin-like growth factor-1 and cognition in normoglycemia, prediabetes, and type 2 diabetes mellitus.

Increased Osteoblast G&#x3b1;S Promotes Ossification by Suppressing Cartilage and Enhancing Callus Mineralization During Fracture Repair in Mice.

&#x3b2;-Arrestin 2 knockout prevents bone loss in response to continuous parathyroid hormone stimulation in male and female mice.

Increased osteoblast G&#x3b1;S level determines bone response to hyperparathyroidism in female mice.

Sex Differences in Metabolic and Behavioral Responses to Exercise but Not Exogenous Osteocalcin Treatment in Mice Fed a High Fat Diet.

Serum osteocalcin is associated with subjective stress in people with depression and type 2 diabetes.

Endostatin as a Mediator Between Endothelial Function and Cognitive Performance in Those at Risk for Vascular Cognitive Impairment.

Growth Hormone Increases Bone Toughness and Decreases Muscle Inflammation in Glucocorticoid-Treated Mdx Mice, Model of Duchenne Muscular Dystrophy.

Intermittent PTH treatment improves bone and muscle in glucocorticoid treated Mdx mice: A model of Duchenne Muscular Dystrophy.

Molecular mechanisms of invertebrate visual systems

Frequent collaborators

Walter Swardfager

Adele Changoor

Marc Grynpas

Nathan Herrmann

Jennifer Harrington

Jeremy Gilbert

Increased osteoblast Gα11 level compromises bone healing quality by suppressing high-density bone formation.

Increased Osteoblast GαS Promotes Ossification by Suppressing Cartilage and Enhancing Callus Mineralization During Fracture Repair in Mice.

β-Arrestin 2 knockout prevents bone loss in response to continuous parathyroid hormone stimulation in male and female mice.

Increased osteoblast GαS level determines bone response to hyperparathyroidism in female mice.