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Research
Latest papers
IGF-I stimulates CCN5/WISP2 gene expression in pancreatic β-cells, which promotes cell proliferation and survival against streptozotocin.
Endocrinology · 2014
Pancreatic islet-specific overexpression of Reg3β protein induced the expression of pro-islet genes and protected the mice against streptozotocin-induced diabetes mellitus.
American journal of physiology. Endocrinology and metabolism · 2011
Latest funding
- $225,000
Metabolic deceleration as a mechanism for the anti-diabetic effects of metformin and berberine
CIHR · 2012 · Principal investigator
2 publications.
IGF-I stimulates CCN5/WISP2 gene expression in pancreatic β-cells, which promotes cell proliferation and survival against streptozotocin.
Chowdhury S, Wang X, Srikant CB, Li Q, Fu M, Gong YJ, Ning G, Liu JL
Pancreatic islet-specific overexpression of Reg3β protein induced the expression of pro-islet genes and protected the mice against streptozotocin-induced diabetes mellitus.
Xiong X, Wang X, Li B, Chowdhury S, Lu Y, Srikant CB, Ning G, Liu JL
Metabolic deceleration as a mechanism for the anti-diabetic effects of metformin and berberine
Principal investigators: Prentki, Marc; Ning, Guang
Keywords: Berberine; Détoxification; Fatty Acids; Glucolipotoxicity; Lipolysis; Metabolic Deceleration; Metformin; Mitochondrial Complex-I; Pancreatic Beta Cell; Type 2 Diabetes
From CIHR, NSERC and SSHRC funding decisions: CIHR since 2008, NSERC since 1991 and SSHRC since 1998, including their latest published competition results.
Frequent collaborators
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