Faculty profile

Marie Trudel

ResearcherMedicineUniversité de Montréal

This profile is built from public research funding records (CIHR, NSERC and SSHRC) and PubMed. We have not imported them from a Université de Montréal directory, so their courses and email address may be missing. Find their university profile.

Marie Trudel

Researcher

Medicine

Health Sciences

Université de Montréal

Montreal, Quebec, Canada

Supervision signals

Has led research grants, which requires an independent research appointment. Ask their department whether they supervise graduate students.
1 publication in the last five years, latest in 2023.
Senior (last) author on 1 recent paper, a sign of leading a research group.
Principal investigator on 2 research grants (2 CIHR), most recently in 2018.
Supervised 3 trainees who won CIHR awards, most recently in 2011.

These signals come from public records, including CIHR, NSERC and SSHRC funding decisions. Ask the professor or the graduate program whether they are taking new students.

Academic metrics

Publications

Citations

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Collaborators

Grants

Publications by year

Counts cover the works listed on the university profile; citation counts come from Dimensions.

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Signals and details

Research

Cystic Disease Epithelial Cell Signaling Genetic Mouse Model Polycystic Kidney Disease Gene Therapy Regulation Hemoglobinopathies Molecular Genetics Sickle Cell Disease

Latest papers

Modeling Pkd1 gene-targeted strategies for correction of polycystic kidney disease.
Molecular therapy. Methods & clinical development · 2023 · senior author
Trpv1 and Trpa1 are not essential for Psickle-like activity in red cells of the SAD mouse model of sickle cell disease.
Blood cells, molecules & diseases · 2021
Corrigendum to "Combined genetic disruption of K-Cl cotransporters and Gardos channel KCNN4 rescues erythrocyte dehydration in the SAD mouse model of sickle cell disease" [Blood Cells Mol. Dis. (2019) start page-end page not yet assigned] https://doi.org/10.1016/j.bcmd.2019.102346.
Blood cells, molecules & diseases · 2020

All 29 publications

Latest funding

Targeting Kcnn4 in polycystic kidney disease : a preclinical mechanistic proof of concept study
CIHR · 2018 · Nominated PI
$933,300
Understanding transcriptional and epigenetic control by Gfi1b towards the development of a therapy for sickle cell disease
CIHR · 2014 · Nominated PI
Polycystic kidney disease pathogenic mechanism: analysis of the role of KCNN4 in orthologous mouse models.
CIHR · 2011 · Supervisor
$17,500

All 5 grants and awards

29 publications.

Modeling Pkd1 gene-targeted strategies for correction of polycystic kidney disease.

Kurbegovic A, Pacis RC, Trudel M

Molecular therapy. Methods & clinical development2023Senior author

DOI: 10.1016/j.omtm.2023.03.016 PMID: 37214311

Trpv1 and Trpa1 are not essential for Psickle-like activity in red cells of the SAD mouse model of sickle cell disease.

Vandorpe DH, Rivera A, Shmukler BE, Wohlgemuth JG, Dlott JS, Snyder LM, Trudel M, Brugnara C, Alper SL

Blood cells, molecules & diseases2021

DOI: 10.1016/j.bcmd.2021.102619 PMID: 34768199

Corrigendum to "Combined genetic disruption of K-Cl cotransporters and Gardos channel KCNN4 rescues erythrocyte dehydration in the SAD mouse model of sickle cell disease" [Blood Cells Mol. Dis. (2019) start page-end page not yet assigned] https://doi.org/10.1016/j.bcmd.2019.102346.

Shmukler BE, Rivera A, Bhargava P, Nishimura K, Hsu A, Kim EH, Trudel M, Rust MB, Hubner CA, Brugnara C, Alper SL

Blood cells, molecules & diseases2020

DOI: 10.1016/j.bcmd.2019.102390 PMID: 31931456

Combined genetic disruption of K-Cl cotransporters and Gardos channel KCNN4 rescues erythrocyte dehydration in the SAD mouse model of sickle cell disease.

Shmukler BE, Rivera A, Bhargava P, Nishimura K, Hsu A, Kim EH, Trudel M, Rust MB, Hubner CA, Brugnara C, Alper SL

Blood cells, molecules & diseases2019

DOI: 10.1016/j.bcmd.2019.102346 PMID: 31352162

c-Myc is a regulator of the PKD1 gene and PC1-induced pathogenesis.

Parrot C, Kurbegovic A, Yao G, Couillard M, Côté O, Trudel M

Human molecular genetics2019Senior author

DOI: 10.1093/hmg/ddy379 PMID: 30388220

Casein kinase 1ε and 1α as novel players in polycystic kidney disease and mechanistic targets for (R)-roscovitine and (S)-CR8.

Billot K, Coquil C, Villiers B, Josselin-Foll B, Desban N, Delehouzé C, Oumata N, Le Meur Y, Boletta A, Weimbs T, Grosch M, Witzgall R, Saunier S, Fischer E, Pontoglio M, Fautrel A, Mrug M, Wallace D, Tran PV, Trudel M, Bukanov N, Ibraghimov-Beskrovnaya O, Meijer L

American journal of physiology. Renal physiology2018

DOI: 10.1152/ajprenal.00489.2017 PMID: 29537311

Approaches for Analysis of Erythroid Cell Parameters and Hemoglobinopathies in Mouse Models.

Trudel M, Sedzro JC

Methods in molecular biology (Clifton, N.J.)2018First author

DOI: 10.1007/978-1-4939-7428-3_6 PMID: 29076086

Cardiorespiratory pathogenesis of sickle cell disease in a mouse model.

Ren J, Ding X, Trudel M, Greer JJ, MacLean JE

Scientific reports2017

DOI: 10.1038/s41598-017-08860-8 PMID: 28819305

Genome-wide association study of erythrocyte density in sickle cell disease patients.

Ilboudo Y, Bartolucci P, Rivera A, Sedzro JC, Beaudoin M, Trudel M, Alper SL, Brugnara C, Galactéros F, Lettre G

Blood cells, molecules & diseases2017

DOI: 10.1016/j.bcmd.2017.05.005 PMID: 28552477

microRNA-17 family promotes polycystic kidney disease progression through modulation of mitochondrial metabolism.

Hajarnis S, Lakhia R, Yheskel M, Williams D, Sorourian M, Liu X, Aboudehen K, Zhang S, Kersjes K, Galasso R, Li J, Kaimal V, Lockton S, Davis S, Flaten A, Johnson JA, Holland WL, Kusminski CM, Scherer PE, Harris PC, Trudel M, Wallace DP, Igarashi P, Lee EC, Androsavich JR, Patel V

Nature communications2017

DOI: 10.1038/ncomms14395 PMID: 28205547

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Marie Trudel

Research

Latest papers

Latest funding

Modeling Pkd1 gene-targeted strategies for correction of polycystic kidney disease.

Trpv1 and Trpa1 are not essential for Psickle-like activity in red cells of the SAD mouse model of sickle cell disease.

Corrigendum to "Combined genetic disruption of K-Cl cotransporters and Gardos channel KCNN4 rescues erythrocyte dehydration in the SAD mouse model of sickle cell disease" [Blood Cells Mol. Dis. (2019) start page-end page not yet assigned] https://doi.org/10.1016/j.bcmd.2019.102346.

Combined genetic disruption of K-Cl cotransporters and Gardos channel KCNN4 rescues erythrocyte dehydration in the SAD mouse model of sickle cell disease.

c-Myc is a regulator of the PKD1 gene and PC1-induced pathogenesis.

Casein kinase 1ε and 1α as novel players in polycystic kidney disease and mechanistic targets for (R)-roscovitine and (S)-CR8.

Approaches for Analysis of Erythroid Cell Parameters and Hemoglobinopathies in Mouse Models.

Cardiorespiratory pathogenesis of sickle cell disease in a mouse model.

Genome-wide association study of erythrocyte density in sickle cell disease patients.

microRNA-17 family promotes polycystic kidney disease progression through modulation of mitochondrial metabolism.

Targeting Kcnn4 in polycystic kidney disease : a preclinical mechanistic proof of concept study

Understanding transcriptional and epigenetic control by Gfi1b towards the development of a therapy for sickle cell disease

Polycystic kidney disease pathogenic mechanism: analysis of the role of KCNN4 in orthologous mouse models.

Characterization of developmental regulatory interactions in fetal to adult globin switch

Functional characterization of polycystin-1 extracellular domain

Frequent collaborators

Guy Cloutier

Eric Milot

Guillaume Lettre

Maxime Bouchard

Elliot Drobetsky

Alain Nepveu

Marie Trudel

Research

Latest papers

Latest funding

Modeling Pkd1 gene-targeted strategies for correction of polycystic kidney disease.

Trpv1 and Trpa1 are not essential for Psickle-like activity in red cells of the SAD mouse model of sickle cell disease.

Corrigendum to "Combined genetic disruption of K-Cl cotransporters and Gardos channel KCNN4 rescues erythrocyte dehydration in the SAD mouse model of sickle cell disease" [Blood Cells Mol. Dis. (2019) start page-end page not yet assigned] https://doi.org/10.1016/j.bcmd.2019.102346.

Combined genetic disruption of K-Cl cotransporters and Gardos channel KCNN4 rescues erythrocyte dehydration in the SAD mouse model of sickle cell disease.

c-Myc is a regulator of the PKD1 gene and PC1-induced pathogenesis.

Casein kinase 1&#x3b5; and 1&#x3b1; as novel players in polycystic kidney disease and mechanistic targets for (R)-roscovitine and (S)-CR8.

Approaches for Analysis of Erythroid Cell Parameters and Hemoglobinopathies in Mouse Models.

Cardiorespiratory pathogenesis of sickle cell disease in a mouse model.

Genome-wide association study of erythrocyte density in sickle cell disease patients.

microRNA-17 family promotes polycystic kidney disease progression through modulation of mitochondrial metabolism.

Targeting Kcnn4 in polycystic kidney disease : a preclinical mechanistic proof of concept study

Understanding transcriptional and epigenetic control by Gfi1b towards the development of a therapy for sickle cell disease

Polycystic kidney disease pathogenic mechanism: analysis of the role of KCNN4 in orthologous mouse models.

Characterization of developmental regulatory interactions in fetal to adult globin switch

Functional characterization of polycystin-1 extracellular domain

Frequent collaborators

Guy Cloutier

Eric Milot

Guillaume Lettre

Maxime Bouchard

Elliot Drobetsky

Alain Nepveu

Casein kinase 1ε and 1α as novel players in polycystic kidney disease and mechanistic targets for (R)-roscovitine and (S)-CR8.